[1] GOZAL E, ORTIZ L A, ZOU X, et al. Silica-induced apoptosis in murine macrophage Involvement of tumor Necrosis Factor-α and Nuclear Factor-ΚB Activation[J]. Am J Respir Cell Mol Biol, 2002, 27(1):91-98.
[2] PORTER D W, MILLECCHIA L L, WILLARD P, et al. Nitric oxide and reactive oxygen species production causes progressive damage in rats after cessation of silica inhalation[J].Toxic Sci,2006,90(1):188-197.
[3] 徐勤惠,杨平平,张天裘,等.巨噬细胞内溶菌酶活力测定方法[J].中国实验临床免疫学杂志,1992,4(4):1-4.
[4] 雷曙光,罗永煌,陈君.IND研究概况及其在动物上的应用[J].中国动物保健,2011,13(1):14-17
[5] ERDOGDU G, HASIRCI V. An overview of the role of mineral solubility in silicosis and asbestosis[J].Environ Res,1998,78(1):38-42.
[6] RASMUSSEN M. Treatment of elevated intracranial pressure with indomethacin: friend or foe[J]. Acta Anaesthesiol scand, 2005,49(3):341-350.
[7] 孙玮玮,宋志芳,曾昭玉.染石英大鼠肺泡巨噬细胞溶菌酶表达的研究[J].中华劳动卫生职业病杂志,2006,24(9):558-559.
[8] MYRVIK Q,LEAKE E S,FARISS B. Studies on pulmonary alveolar macrophages from the normal rabbit: a technique to procure them in a high states of purity[J], J Immol,1961,86(2):128-132.
[9] 史景熙,潘璠.矽肺肺泡巨噬细胞溶菌酶的促成纤维细胞增殖作用[J].中央民族大学学报(自然科学版),2004,13(4):347-359,379.
[10] MIGLIACCIO C T, HAMILTON R F, HOLIAN A. Increase in a distinct pulmonary macrophage subset possessing an antigen-presenting cell phenotype and in vitro APC activity following silica exposure[J], Toxicol Appl Pharmacol,2005,205(2):168-176.
[11] THIBODEAU M S, GIARDINA C, KNECHT D A, et al. Silica-induced apoptosis in mouse alveolar macrophages is initiated by lysosomal enzyme activity[J]. Toxicol Sci,2004,80(1):34-48.
[12] 张明君,杨旅军,谢思田,等.儿童烧伤治疗中复合溶菌酶的临床应用[J].中国医药指南,2013,11(19):411-412.
[13] 董士华,曲狄.复合溶菌酶治疗儿童烧伤的临床效果分析[J].中国医药导刊,2014,16(5):878-879. |
